DepartmentTemple University Abroad. Rome Campus. Gallery of Art
Africa--Emigration and immigration--Exhibitions
Europe--Emigration and immigration--Exhibitions
Europe--Emigration and immigration--Government policy--Exhibitions
Permanent link to this recordhttp://hdl.handle.net/20.500.12613/171
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Abstract"This project, and its resulting exhibition, which includes text and images, is dedicated to those souls who are navigating the Sahara as well as those that have been lost among its dunes where their bodies 'instantly mummified'. Throughout, the intent of the project has been to identify traces of these migrants' journey from Africa into Europe; through data and policy analysis which are woven together with the images and texts so as to relate fragmentary memories of the journey. Given that words cannot express certain elements of the migratory condition, and statistical data can only represent the aggregations and that which is recorded, the images help provide the deeper insights that enable us to decipher such a complex phenomenon. Recognizing the imperative to sketch out a "new politics of truth, one founded in contingency and self-transformation," it is our aim to move away from a conventional interpretation of migration, which relegates the discussion to the dim space between illegality and victimhood."--Lorenzo Rinelli's academia.edu webpage.
DescriptionPublished to accompany the exhibition, Safe Travels/Interrupted Flows, curated by Marta Bordignon, Camilla Lai, and Lorenzo Rinelli in collaboration with Shara Wasserman. Held June 4-24, 2019 at the Gallery of Art, Temple University Rome.
CitationBordignon, M. & Rinelli, L. (2019). Interrupted Flows. Temple University Rome.
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Showing items related by title, author, creator and subject.
Re-envisioning the 1876 Centennial Exhibition: New Exhibit Solutions for an Old Interpretive ProblemBruggeman, Seth C.; Winling, LaDale (Temple University. Libraries, 2011)This paper takes a fresh look at the Centennial Exhibition of 1876 and exhibits that interpret it, and suggests new exhibit strategies to re-interpret this complicated moment in American history.
Exhibiting Evangelicalism: Commemoration, Conservative Christianity, and Religion's Presence of the PastBruggeman, Seth C.; Lowe, Hilary I.; Berman, Lila Corwin; Linenthal, Edward Tabor (Temple University. Libraries, 2020)“Exhibiting Evangelicalism” is a history of evangelical historical museums in the United States. It argues that conservative Protestant Christians in the United States developed practices for preserving and interpreting the past in public and deployed those practices toward varying theological, cultural, and political ends—an approach I term “evangelical heritage.” It further contends that evangelical heritage performed important work for its purveyors. Amid the boom in church attendance and religious affiliation after World War II, conservative Protestants deployed evangelical heritage to forge what they termed “neo-evangelicalism,” a rebranding of the old-time religion for postwar society. They also engaged evangelical heritage in their crusade to “win America for Christ,” convinced that an encounter with their tradition’s proud past could entice outsiders to convert to Christian faith. These elements never fully disappeared from the function of evangelical heritage. Even so, evangelical heritage did change over time. During the national bicentennial, for instance, evangelical heritage became a means by which neo-evangelicals, internally divided over matters of faith and politics, could project a united front by mapping their proud past onto the nation’s history. Such optimism did not last long. As the national consensus about the past shattered in the 1970s and 1980s, evangelical heritage morphed yet again. By the twenty-first century it had become a vehicle for nostalgia, immersing visitors in a mythic past that offered an imagined sense of comfort and reassurance amid conservative Protestants’ perceived loss of political and social influence. Evangelical heritage did not develop and evolve in a vacuum, however. From the start, it existed within and contributed to broader patterns of historical commemoration. In the postwar era, for instance, experiments in evangelical heritage intersected and overlapped with discourses and practices among bureaucrats, business leaders, social reformers, heritage professionals, and others regarding historic preservation, urban renewal, and the political purposes of civic memory. In the 1970s, neo-evangelical museum-makers helped to invent public history’s turn toward emotion, immersion, and experience as techniques through which to build visitors’ historical knowledge. As that trend became subject to intense internecine debate among public history professionals in the 1980s and 1990s, some conservative Protestant commemorators turned away from the mainstream of public history discourse. Instead, they embraced the theme park as a means of conveying ideological authority while retaining the trappings of the traditional museum as a way of courting intellectual authority—a trend that reached its apex at the turn of the twenty-first century.
MUTANT DROSOPHILA LACKING SIALIC ACID EXHIBIT NEURODEGENERATION AND METABOLIC DYSFUNCTION LEADING TO A DIABETIC STATEPalter, Karen; Gruberg, Edward R.; Waring, Richard B.; Betenbaugh, Michael (Temple University. Libraries, 2012)Sialylation, a posttranslational modification of both glycolipids and glycoproteins, is typically found on the terminal positions of glycan chains. Unique among most other sugars, sialic acids are nine carbon sugars that are negatively charged and undergo a variety of side group modifications, which contribute to its role in cell-cell interactions and receptor recognition. While the majority of Drosophila glycoproteins do not terminate in sialic acid compared to mammalian glycoproteins, a sialic acid synthetic pathway is present in Drosophila but it is developmentally regulated and appears to be restricted to the nervous system (Kim et al., 2002; Koles et al., 2004). In order to investigate the role of the sialic acid pathway in Drosophila, we generated a null mutation of the sialic acid synthase gene (SAS) by imprecise excision of a nearby transposable element. Homozygous null flies exhibit partial lethality, male sterility and undergo age dependent neurodegeneration as evidenced by loss of locomotion and increased vacuolization in the brain. Mutant flies also have a shortened life span and display increased sensitivity to heat as they age. To identify protein targets of sialylation that possibly contributed to these phenotypes, a very sensitive solid-phase extraction method was used to capture sialylated glycopeptides from head extracts of wild type and SAS null flies. In collaboration with M. Betenbaugh and H. Zhang at Johns Hopkins University, I identified three sialylated peptides; the major peptide target was derived from the Shaker voltage-dependent potassium channel. The other two peptides were encoded by genes of unknown function. Electrophysiological measurements performed on control and SAS mutant larvae at the pre- and post-synaptic larval neuromuscular junction (in collaboration with T. Dean and A. Seghal, University of Pennsylvania) showed that loss of sialylation induced a depolarizing shift in the gating parameters of the Shaker ion channels, similar to what was previously reported in mammalian cell culture (Johnson and Bennett, 2007). Pre-synaptic neurons from the mutants displayed a two to three fold increase in the number of miniature excitatory peaks suggesting that the neurons were hyperactive. Since Shaker is a major target of sialylation in brain neurons, I suggest that the loss of sialylation of Shaker plays a major role in the neurodegeneration phenotype observed in our SAS null mutant flies. SAS mutant flies are unusually sensitive to starvation, typical of flies that cannot maintain metabolic homeostasis. Upon 24 h of starvation, mutant flies differed from control flies in that they consumed most of their triglyceride stocks, they displayed poor locomotion, had smaller sized cells in their fat body, and reduced their glycogen stores significantly. Mutant flies expressed and likely secreted an excess of insulin like protein, hyperinsulinemia, for the first five days after eclosion. However, as the flies aged (14-21 days) they had high hemolymph sugar, low insulin like protein expression and fewer number of insulin producing cells. All these phenotypes are similar to diabetic patients, as diabetic patients also have metabolic inflexibility, high blood sugar, low insulin secretion, and fewer number of insulin producing cells. It is now known that the Shaker homolog KV1 is expressed in human pancreatic β cells which secrete insulin (Ma et al., 2011). I propose that in our mutants the failure to sialylate the Shaker channel, which is known to be present in insulin producing cells (IPC ), will result in those cells secreting an excess of insulin , which in turn causes the metabolic defects leading to a diabetic state. By studying our sialic acid null mutants we can obtain useful information about how diabetes develop. Using the powerful genetics of flies, we can perform screens to identify novel genes that either enhance or reduce the sensitivity to starvation of our SAS mutants and thus play a role in the development or potential treatment of diabetes